NAD^+ can be regenerated in the cytoplasm if NADH rears with any of the followin

Question

NAD^+ can be regenerated in the cytoplasm if NADH rears with any of the following pyruvate. Glucokinase has greater than the normal blood glucoses concentration. in in mused. is inhibited by glucose 6-phosphate. is also known as the the GLUT-2 protean. has glucose 6-phosphacase activity as well as kinase activity. 6-Phosphofracao 1 kinase activity can decreased by all of the following ATP at high concentrations citrate. AMP. low pH. decreased concentration of fructose 2, 6-bisphosphase. Which of the following spores gluconeogenesis alpha + aspartame pyruvate + ATP + HCO ADP + P_1 H^+ acryl CoA + H_2O + CoA lupine lysine degradation. In the Cori cycle. only reissues with aerobic metabolism (i, e., mitochondria and O_2) are involved. a three- carbon compound arising from glycolysis is converted to glucose at the expense of energy from fatty acid aviation. glucose is converged to pyruvate in anaerobic and this pyruvate returns to the liver, where it is converted to glucose. the same amount of ATP is used in the liver to synthesize glucose as is released during glycolysis, leading to no net effect on whole body energy balance. nitrogen from alanine must be converted to urea, increasing the amount of energy required to drive the processes. When blood glucagon rises, which of the following hepatic enzyme activates falls? acetylate cycles. protein kinase. 6-pheaphofrucro-2-kinase, fructose 1, 6-bisphosphacase hexokinase. Questions 7 and 8: Patients with McArdle's discase suffer from painful mused cramps and are unable to perform strenuous exercise. This disease, also called type V glycogen storage disease, is caused by the absence of mused phosphorylase. Unlike patients with Von Gierke's disease (Type I glycogen storage disease caused by a deficiency of glucose 6-ptaptutase), McArdle's patients do not typically suffer hypoglycemia on fasting. Von Gierke's patients show lactic acidemia, but McArdle's patients fail to demonstrate lactic academia following exercise. McArdle's patients" fail to glycogen appropriately. do not respond to glucagon in a normal fashion. show a rescued skate of glycolysis daring have an impaired cricarborylic acid cycle. have reduced lactic acid because all of the lactase is pyruvate. Glucose 6-phosphacase, which is deficient in Von necessary for the production of blood glucose from: liver glycogen. fructose. amino acid carbon chains. lactose. all of the above. Questions 9 and 10 Malignant hypothermia is a in which exposure to certain agent, especially the widely used anesthetic halothane, produces a dramatic rite in body hypothermia, and mused rigidity, Death is rapid if the underused and may occur the first time a susceptible person is The defies causes an inappropriate release of Ca^2+ from the reticulum of Many heat -producing procures are an uncontrolled fashion by the release of Ca^2+, including glycolysis glycogenesis. Ca^2+ increases glycogenesis by: activating phosphorylate kinase b, even in the absence of binding to phosphorylase b. activating phosphoprocein phosphatase. inhibiting phosphoprocein phosphatase. prosecuting cAMP from degradation. Phosphorylation - dephosphorylation and allow Eric enaymes play roles in stimulating glycogen degradation. All following result in enzyme activation except: phosphorylation of phosphorylase kinase. binding of AMP to phosphorylase b. phosphorylation of phosphorylase phosphorylation of protein kinase A. dephosphorylation of glycogen synthase. Patients with hereditary fructose from of the enzyme aldoses. Consumption of ATP and P, in the liver, which in turn Much of the cell damage can be attributed to the maintain normal ion gradients by ATP-dependent pumps. The first step in liver's metabolism of fructose is: isomerization to glucose. phosphorylation to fructose 1, 6-bisphosphate by ATP. phosphorylation to fructose 1-bisphosphate by ATP. damage by aldoses. The products initially produced by aldoses action on the formed from fructose are. two molecules of dihydroxy acetone phosphate. two molecules of glyceraldehyde 3-phosphat. two molecules of lactate. dihydroxyacetone phosphate and glyceraldehyde 3-phosphate. dihydroxyacetone phosphate and glyceraldehyde.

Explanation / Answer

Q1.

NAD+ can be regenerated in the cytoplasm if NADH reacts with any of the following except:

A) pyruvate --> correct, via the lactate reaction of pyruvate

b) dihydroxyacetone phosphate --> Correct, via the Glycerol phosphate shuttle

c) oxaloacetate --> Correct, via the Malate dehydrogenase enzyme. It oxidizes from malate to oxaloacetate using the reduction of NAD+ to NADH

d) the flavio boundto NADH dehdrogenase --> this must be false. No data is known to form NADH via flavions

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