19. How do white blood cells internalize bacteria? B) Phagocytosis C) D) Recepto

Question

19. How do white blood cells internalize bacteria? B) Phagocytosis C) D) Receptor-mediated endocytosis E) Any type of endocytosis 20 synthetic compounds that replicate the actvity of natural messengers by binding to receptors are known as A) agonists. B) receptor upregulators. C) specific proteoglycans. ecans. E) antagonists. help turn proteins "on and off through changes in phosphorylation status. B) are enzymes that destroy damaged proteins. C) sense short poly-A tails in mRNAs as a signal to degrade them. Dl destroy second messengers, thereby turning off a signal transduction pathway. 21. Kinases and phosphatases are essential in cells because they E) all of the above 22. You have recently identified a molecule that you believe to be a signal transduction pathway's ligand. All you know about this ligand is that chemically it is hydrophilic. As a result, you expect it to interact with its receptor A) at the outer cell surface. B) within the cytoplasm of the cell. C) within the nucleus of the cell. D) on the cytoplasmic side of the plasma membrane. E) all of the above 23. technique that could be used to demonstrate that Glut1 and Glut3 are overexpressed in cancer cells as compared to normal cells is A) DNA sequencing B) Mass spectrometry C) Western blotting D) Thin layer chromatography E) Electron microscopy 24. Allosteric enzyme inhibitors are a) Likely to compete with the substrate for the active site b) Can be competed out by excess substrate c) Bind to site distinct from the active site d) Cannot be competed out by excess substrate e) Both choices C and D

Explanation / Answer

19.B) phagocytosis.

20.A) agonists

21.A) Kinase and phosphatase are important as they help to turn proteins on and off through changes in the phophorylation status.

22.a) at the outer surface

23.C) western blotting.

24c) bind to the site distinct from the active site.

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