Explain why glucose consumption roust increase in hypoxic (oxygen deficient) tis

Question

Explain why glucose consumption roust increase in hypoxic (oxygen deficient) tissues to provide the same amount of ATP that could be produced from glucose in normoxic (normal O_2 levels) tissues. A. Oxygen is the limiting reactant in ATP producing, so ATP is produced in hypoxic tissues slower than in normoxic tissues. B. In hypoxic tissues, ATP is produced only by glycolysis, whereas in normoxic tissues, the citric acid cycle + oxidative phosphorylation can be used. C. In hypoxic tissues, only by the citric acid cycle is responsible for ATP production Which of the following is NOT an electron acceptor in the mitochondrial respiratory chain? A. FMN B. FAD C. Fe^3+ D. Cu^2+ E.COQH_2

Explanation / Answer

6. Explain why glucose consumption must increase in hypoxic (oxygen deficient) tissues to provide the same amount of ATP that could be produced from glucose in normoxic (normal oz levels) tissues.

A oxygen is the limiting reactant in ATP producing, so ATP is produced in hypoxic tissues slower in normoxic tissues

. B. In hypoxic tissues, ATP is produced only by glycolysis, whereas in normoxic tissues, the citric acid cycle oxidative phosphorylation can be used.-ANSWER

-e In hypoxic tissues, only by the citric acid cycle is responsible for ATP production

17. Which of the following is NOT an electron acceptor in the mitochondrial respiratory chain?

A. FMN

B. FAD

C. Fe

D. Cu -ANSWER

E. CoQH

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[FMN] accepts electron pair from NADH

FAD + 2H+ + 2e- g FADH2

cytochrome c, Fe3+  + e- g cytochrome c, Fe2

  CoQ (UQ) + 2 H+ + 2 e- g CoQH2 (UQH2),              

Thus, CoQ is the electron acceptor and NADH is the electron donoR

Hypoxia decreases ATP generation to a limited extent by bringing down the action of the electron transport chain through actuation of the interpretation calculate hypoxia-inducible component . The decline in electron transport restrains the overproduction of reactove oxygen species amid hypoxia and moderates the rate of oxygen consumption to forestall anoxia. In this audit, we talk about these components that decrease metabolic free market activity for adjustment to hypoxia.

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