A patient experiences a sudden rupture of the chordae tendineae that results in
ID: 3517644 • Letter: A
Question
A patient experiences a sudden rupture of the chordae tendineae that results in the mitral valve not closing properly. In this patient:
(a) Describe the filling of the left ventricle during diastole. (0.5 mark)
(b) Describe the movement of the left ventricular blood during systole. (1 mark)
(c) Describe what may happen to blood pressure in the pulmonary vein and why. (2 marks)
(d) Describe what might be happening in the lungs and the symptoms that may result. (2 marks)
(e) Describe what might be happening to cardiac output and arterial blood pressure both in the short-term and in the long-term. (4.5 marks)
Explanation / Answer
Due to the rupture chordae tendinae of mitral valve there is-mitral valve prolapse and due to the mitral valve prolapse there is mitral regurgitation occurs.
A) the feeling of the left ventricle during the diastole is increased because due to mutual regurgitation the left Atrium volume is increased so when there is diastole there will be more amount of volume entering into the left ventricle from left Atrium. Show volume overload occur in the left ventricle during diastole.
B) during the systole left ventricle pump more amount of blood into the aorta and previously compromised blood into a aorta is becomes normal, but it produce left ventricular hypertrophy .
C) in the chronic dysfunction phase of the middle regurgitation there isp muscle dysfunction of the left ventricular occur which lead to increased left ventricular and left Atrium pressure which ultimately lead to increase p back pressure into pulmonary circuit lead to increase pressure in to pulmonary vein and pulmonary oedema develop.
D) in the lungs the pulmonary edema develop and which for the lead to cardiogenic shock due to development of pulmonary edema there will be dysania inspiratory Crackles and cough From lungs.
E) in the short term there is chronic compensated mitral valve regurgitation there will be accommodation of Axis volume by ecentric left ventricular hypertrophy and finally it normal the stroke volume and cardiac output .
In the chronic decompensated phase of mitral regurgitation there will be failure of left ventricular muscle and increased left ventricular and atrial pressure lead to development of pulmonary edema and finally cardiogenic shock.
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