What does this table suggest about the locations (plasmid vs chromosomal) for an
ID: 178445 • Letter: W
Question
What does this table suggest about the locations (plasmid vs chromosomal) for antibiotic resistance, and the genes for virulence factors P fimbriae and hemolysin? Does this make sense? Why?
I DON'T HAVE THE TABLE, HOWEVER, IF YOU CAN MAKE ANY INFERENCES FROM THIS INFORMATION THAT WOULD BE GREAT!
1. Table 2 shows the relationship between aerobactin genotype and other metrics. For example: 11 of 13 (85%) of E. coli strains that did not have aerobactin genes were found in compromised patients, while 36 of 45 (80%) of E. coli strains that had aerobactin genes were found in these patients. Therefore, aerobactin genes do not influence whether E. coli is a problem for compromised patients. On the other hand, of the 45 strains that had the genes, 12 of 12 (100%) that had plasmid-encoded genes were found in compromised patients, but only 24 of 33 (73%) that had chromosome-encoded genes were found in compromised patients. Therefore, plasmid-encoded aerobactin is associated with infections of compromised patients.Explanation / Answer
Believe the table suggest that the genes encoded in plasmid has high virulence factor that causes infection since 100% of the starins were found in infected compromised individuals and only 73% of strains were found to be chromosome encoded genes which caused infections in compromised individuals.
Hence it is fine that plasmid encoded genes causes high virulence.
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